eeg amplifier active two Search Results


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brain products gmbh two 32-channel, nonmagnetic, battery-operated electroencephalographic amplifiers (brainamp mr)
Two 32 Channel, Nonmagnetic, Battery Operated Electroencephalographic Amplifiers (Brainamp Mr), supplied by brain products gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/two 32-channel, nonmagnetic, battery-operated electroencephalographic amplifiers (brainamp mr)/product/brain products gmbh
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two 32-channel, nonmagnetic, battery-operated electroencephalographic amplifiers (brainamp mr) - by Bioz Stars, 2026-06
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Alpha-Omega Engineering eeg/emg activity was amplified (500x), filtered (1 – 100 hz for eeg
Eeg/Emg Activity Was Amplified (500x), Filtered (1 – 100 Hz For Eeg, supplied by Alpha-Omega Engineering, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
eeg/emg activity was amplified (500x), filtered (1 – 100 hz for eeg - by Bioz Stars, 2026-06
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Pinnacle Technology Inc two eeg channel, two emg channel mouse pre-amplifier
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Two Eeg Channel, Two Emg Channel Mouse Pre Amplifier, supplied by Pinnacle Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/two eeg channel, two emg channel mouse pre-amplifier/product/Pinnacle Technology Inc
Average 90 stars, based on 1 article reviews
two eeg channel, two emg channel mouse pre-amplifier - by Bioz Stars, 2026-06
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BioSemi dual eeg active two
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Dual Eeg Active Two, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
dual eeg active two - by Bioz Stars, 2026-06
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BioSemi high density eeg active two recording device
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
High Density Eeg Active Two Recording Device, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/high density eeg active two recording device/product/BioSemi
Average 90 stars, based on 1 article reviews
high density eeg active two recording device - by Bioz Stars, 2026-06
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BioSemi eeg electrodes active ii
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Eeg Electrodes Active Ii, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/eeg electrodes active ii/product/BioSemi
Average 90 stars, based on 1 article reviews
eeg electrodes active ii - by Bioz Stars, 2026-06
90/100 stars
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brain products gmbh two brainamp mr plus eeg amplifiers
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Two Brainamp Mr Plus Eeg Amplifiers, supplied by brain products gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/two brainamp mr plus eeg amplifiers/product/brain products gmbh
Average 90 stars, based on 1 article reviews
two brainamp mr plus eeg amplifiers - by Bioz Stars, 2026-06
90/100 stars
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90
BioSemi active two eeg system with 65 ag/agcl electrodes
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Active Two Eeg System With 65 Ag/Agcl Electrodes, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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active two eeg system with 65 ag/agcl electrodes - by Bioz Stars, 2026-06
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BioSemi 64+8 channel surface electroencephalography (eeg) system active-two
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
64+8 Channel Surface Electroencephalography (Eeg) System Active Two, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
64+8 channel surface electroencephalography (eeg) system active-two - by Bioz Stars, 2026-06
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BioSemi active two eeg system’s analog input box
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Active Two Eeg System’s Analog Input Box, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
active two eeg system’s analog input box - by Bioz Stars, 2026-06
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brain products gmbh two multi-channel eeg amplifiers
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Two Multi Channel Eeg Amplifiers, supplied by brain products gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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two multi-channel eeg amplifiers - by Bioz Stars, 2026-06
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BioSemi dual-eeg recording setup consisting of two biosemi active two systems
Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , <t>spectrograms,</t> <t>EEG/EMG</t> traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .
Dual Eeg Recording Setup Consisting Of Two Biosemi Active Two Systems, supplied by BioSemi, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/dual-eeg recording setup consisting of two biosemi active two systems/product/BioSemi
Average 90 stars, based on 1 article reviews
dual-eeg recording setup consisting of two biosemi active two systems - by Bioz Stars, 2026-06
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Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , spectrograms, EEG/EMG traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .

Journal: bioRxiv

Article Title: A role for thalamic projection GABAergic neurons in circadian responses to light

doi: 10.1101/2022.02.24.481804

Figure Lengend Snippet: Ablation of Sox14 + IGL/LGv neurons causes delayed vigilance state transitions at circadian light changes. A , B , spectrograms, EEG/EMG traces, and hypnograms over a one-hour period from a representative control mouse and an ablated mouse. C , over a 24-hour period control and Sox14 + IGL/LGv-ablated mice spend a similar percentage of time in Wake (48.24 % ± 1.96 % vs 48.22 % ± 1.71 %, p = 0.99), NREM (46.73 % ± 1.80 % vs 46.38 % ± 1.71 %, p = 0.89) and REM (5.03 % ± 0.20 % vs 5.39 % ± 0.17 %, p = 0.2). Values are mean ± s.e.m., t-test. D , E , distribution of Wake, NREM and REM in the two hours preceding and following each light transition. Light and dark hours are shaded in yellow and grey, respectively (see for comprehensive statistics). F , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the light to dark transition. Control group: Wake -1 : 33.17 % ± 2.98 %, Wake 1 : 80.26 % ± 3.49 %, p < 0.0001, Wake 2 : 72.61 % ± 7.76 %, p = 0.001; NREM -1 : 60.08 % ± 2.65 %, NREM 1 : 18.39 % ± 3.29 %, p = 0.0001, NREM 2 : 25.17 % ± 7.04 %, p = 0.0015; REM -1 : 6.75 % ± 0.52 %, REM 1 : 1.35 % ± 0.45 % p = 0.0004, REM 2 : 2.18% ± 0.85 % p = 0.007. Ablated group: Wake -1 36.57 % ± 5.34 %, Wake 1 : 56.67 % ± 3.49 %, p = 0.093, Wake 2 : 75.23 % ± 7.24 %, p = 0.008; NREM -1 56.35 % ± 4.73 %, NREM 1 : 38.19 % ± 7.68 %, p = 0.093, NREM 2 : 22.36 % ± 6.64 %, p = 0.0081; REM -1 : 7.08 % ± 0.78 %, REM 1 : 5.10 % ± 1.16 %, p = 0.296, REM 2 : 2.41 % ± 0.90 %, p = 0.031. Values are mean ± s.e.m., t-test except Control REM -1 versus REM 2 , Ablated REM -1 versus REM 1 and REM 2 Wilcoxon test. G , pairwise comparison in the content of Wake, NREM and REM between the hour preceding and each of the two hours following the dark to light transition. Control group: Wake -1 : 94.77 % ± 3.48 %, Wake 1 : 63.47 % ± 9.42 %, p = 0.015, Wake 2 : 36.11 % ± 9.72 %, p = 0.015; NREM -1 : 5.22 % ± 3.48 %, NREM 1 : 34.87 % ± 8.82 % p = 0.015, NREM 2 : 57.51 % ± 8.86 % p = 0.015; REM -1 : 0.00 % ± 0.00 %, REM 1 : 1.66 % ± 0.64 %, p = 0.125, REM 2 : 6.37 % ± 1.09 %, p = 0.015. Ablated group: Wake -1 79.18 % ± 7.51 %, Wake 1 : 82.47 % ± 7.80 %, p = 0.812, Wake 2 : 39.37 % ± 8.09 %, p = 0.017; NREM -1 20.50 % ± 7.34 %, NREM 1 : 17.25 % ± 7.65 %, p = 0.848, NREM 2 : 56.31 % ± 7.22 %, p = 0.021; REM -1 : 0.31 % ± 0.21 %, REM 1 : 0.27 % ± 0.20 %, p > 0.999, REM 2 : 4.31 % ± 1.34 %, p = 0.0313. Values are mean ± s.e.m., Wilcoxon test except Ablated Wake -1 versus Wake 2 and NREM -1 versus NREM 2 t-test. H , fold change in distance travelled in the light phase (12 hours). Total distance in the dark phase (12 hours) was set at 1. Control group: 1.0 ± 0.14 (dark) versus 0.77 ± 0.26 (light); Ablated group: 1.0 ± 0.17 (dark) versus 0.52 ± 0.82 (light). I , Example trajectories for one control and one ablated mouse in the hour preceding and following the light change. Oval: ROI used for automated tracking; yellow: light, grey: dark. Histograms report the fold change in distance travelled in the hour preceding and following the light change. For the light to dark transition distance in the light was set at 1: Control group 1.0 ± 0.21 (light) 5.59 ± 1.36 (dark), p = 0.0078; Ablated group 1.0 ± 0.17 (light) 2.80 ± 0.68 (dark), p = 0.044. J , For the dark to light transition distance in the dark was set at 1: Control group 1.0 ± 0.15 vs 0.64 ± 0.31, p = 0.148; Ablated group 1.0 ± 0.28 vs 0.92 ± 0.29, p = 0.468. Values are mean ± s.e.m., Wilcoxon test except for Ablated light to dark transition t-test. A summary of statistical tests is available in .

Article Snippet: The EEG/EMG signals were sampled at 250 Hz, amplified 100× and low-pass filtered at 100 Hz using a two EEG channel, two EMG channel mouse pre-amplifier (Pinnacle Technology Inc).

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